KRC-408, a novel c-Met inhibitor, suppresses cell proliferation and angiogenesis of gastric cancer

  • Hong, Sang-Won; 
  • Jung, Kyung-Hee; 
  • Park, Byung Hee; 
  • Zheng, Hong-Mei; 
  • Lee, Hee-Seung; 
  • ... Lee, Jongkook; 
  • 외 4명
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초록

Among many cancer therapeutic targets, c-Met receptor tyrosine kinase has recently given particular attention. This kinase and its ligand, hepatocyte growth factor (HGF), play a central role in cell proliferation and the survival of several human cancers. Thus, we developed KRC-408 as a novel c-Met inhibitor and investigated its anti-cancer effects on human gastric cancer. KRC-408 inhibited the phosphorylation of c-Met and its constitutive downstream effectors such as phosphatidylinositol 3-kinase (PI3K), Akt, Mek, and Erk. This compound was found to exert anti-cancer effects stronger than those of 5-fluorouracil (5-FU) on gastric cancer cells, especially cell lines that overexpressed c-Met. Interestingly, cytotoxicity of KRC-408 was lower than that of 5-FU in normal gastric cells. Apoptosis induced by KRC-408 was accompanied by increased levels of cleaved caspase-3 and PARP as well as DNA condensation and fragmentation. Flow cytometry analysis showed an accumulation of gastric cancer cells in the G2/M phase with concomitant loss of cells in the S phase following treatment with this drug. In the angiogenesis studies, KRC-408 inhibited tube formation and migration of human umbilical vein endothelial cells (HUVECs), and suppressed microvessel sprouting from rat aortic rings ex vivo along with blood vessel formation in a Matrigel plug assay in mice. Results of an in vivo mouse xenograft experiment showed that the administration of KRC-408 significantly delayed tumor growth in a dose-dependent manner, and suppressed Akt and Erk phosphorylation as well CD34 expression in tumor tissues. These findings indicate that KCR-408 may exert anti-tumor effects by directly affecting tumor cell growth or survival via the c-Met receptor tyrosine kinase pathway. We therefore suggest that KRC-408 is a novel therapeutic candidate effective against gastric cancers that overexpress c-Met. Crown Copyright (c) 2013 Published by Elsevier Ireland Ltd. All rights reserved.

키워드

KRC-408; Apoptosis; Angiogenesis; Gastric cancer; TYROSINE KINASE DOMAIN; SOMATIC MUTATIONS; OVEREXPRESSION; CARCINOMAS; GENE; AMPLIFICATION; PROGRESSION; EXPRESSION; SURVIVAL; DOCKING
제목
KRC-408, a novel c-Met inhibitor, suppresses cell proliferation and angiogenesis of gastric cancer
저자
Hong, Sang-Won; Jung, Kyung-Hee; Park, Byung Hee; Zheng, Hong-Mei; Lee, Hee-Seung; Choi, Myung-Joo; Yun, Jeong In; Kang, Nam Sook; Lee, Jongkook; Hong, Soon-Sun
DOI
10.1016/j.canlet.2013.01.015
발행일
2013-05-10
유형
Article
저널명
Cancer Letters
권
332
호
1
페이지
74 ~ 82